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pemt in a sentence

"pemt" in Chinese  
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  • The article about PEMT and this article are virtually the same topic.
  • PEMT deficiency prevents artherosclerosis in mice fed high-fat, high-cholesterol diets.
  • Another possibility is that lack of PEMT in adipose tissue may affect normal fat deposition.
  • This is largely a result of lower levels of VLDL lipids in the PEMT-deficient mice.
  • PEMT modulates levels of blood plasma homocysteine, which is either secreted or converted to methionine or cysteine.
  • PEMT gene expression is regulated by transcription factors including promoter region may increase risk of hepatic steatosis from choline deficiency.
  • PEMT deficiency in mice, genetically induced by PEMT gene knockout, produced minimal effect on PE and PC levels.
  • PEMT deficiency in mice, genetically induced by PEMT gene knockout, produced minimal effect on PE and PC levels.
  • The exact mechanism by which PEMT catalyzes the sequential methylation of PE by three molecules of SAM to form PC remains unknown.
  • Purification of PEMT by Neale D . Ridgway and Dennis E . Vance in 1987 produced an 18.3 kDa protein.
  • It's difficult to see pemt in a sentence .
  • PEMT-deficient mice fed high-fat diets have been shown to resist weight gain and be protected from insulin resistance.
  • In humans it is encoded by the " PEMT " gene within the Smith-Magenis syndrome region on chromosome 17.
  • Subsequent cloning, sequencing, and expression of PEMT cDNA resulted in a 22.3 kDa, 199-amino acid protein.
  • The PEMT enzyme converts phosphatidylethanolamine ( PE ) to phosphatidylcholine ( PC ) via three sequential methylations by S-adenosyl methionine ( SAM ).
  • A Val-to-Met substitution at residue 175, leading to reduced PEMT activity, has been linked to non-alcoholic fatty liver disease.
  • Furthermore, the decreased lipid ( PC ) content in VLDLs causes changes in lipoprotein structure which allow them to be cleared more rapidly in the PEMT-deficient mice.
  • It was concluded that insufficient choline resulted in the lack of weight gain, supported by the fact that PC produced via the PEMT pathway can be used to form choline.
  • The human body can produce choline by methylation of phosphatidylethanolamine by N-methyltranferase ( PEMT ) to form phosphatidylcholine in the liver, or it may be consumed from the diet.
  • This is exemplified by the observation that " Pemt-/-" mice ( mice lacking functional PEMT ) will abort at 9 10 days unless fed supplemental choline.
  • This is exemplified by the observation that " Pemt-/-" mice ( mice lacking functional PEMT ) will abort at 9 10 days unless fed supplemental choline.
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